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1205 primer  (Dow Corning)

 
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    Structured Review

    Dow Corning 1205 primer
    Groups division according to the presence or not of scratches, primer and adhesive
    1205 Primer, supplied by Dow Corning, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primer+1205/1205+primer/pmc03881862-63-10-12
    Average 90 stars, based on 1 article reviews
    1205 primer - by Bioz Stars, 2026-10
    90/100 stars

    Images

    1) Product Images from "Bond strength between acrylic resin and maxillofacial silicone"

    Article Title: Bond strength between acrylic resin and maxillofacial silicone

    Journal: Journal of Applied Oral Science

    doi: 10.1590/S1678-77572012000600010

    Groups division according to the presence or not of scratches, primer and adhesive
    Figure Legend Snippet: Groups division according to the presence or not of scratches, primer and adhesive

    Techniques Used:

    Related Articles

    Viscosity:

    Article Title: Microfluidic device and method for concentrating sample containing cells or viruses and lysing cells or viruses, and method of producing the microfluidic device
    Article Snippet: Dow Corning Primer 1205 was spin coated (500 revolutions per minute (“rpm”), 5 seconds (s); 700 rpm, 10 s) on the silicon wafer as an adhesive, a cation exchange membrane containing —SO3—Na+ group is bonded on the silicon wafer using a roller, and then Dow Corning Primer 1205 was spin coated thereon (500 rpm, 5 s; 1500 rpm, 10 s) to form a first adhesive layer and a second adhesive layer on the cation exchange membrane. .. The Dow Corning Primer 1205 includes a low viscosity, such as water does, at room temperature and has no adhesiveness. .. However, when heat is applied to the Dow Corning Primer 1205, the adhesive is solidified and obtains adhesiveness.

    Adhesive:

    Article Title: Microfluidic device and method for concentrating sample containing cells or viruses and lysing cells or viruses, and method of producing the microfluidic device
    Article Snippet: .. In the illustrated embodiments, Dow Corning Primer 1205 was used as the adhesive. ..

    Article Title: Maxillofacial rehabilitation of a large facial defect resulting from an arteriovenous malformation utilizing a two-piece prosthesis.
    Article Snippet: Large facial defects involving the oral cavity can be difficult to restore prosthetically because of a lack of anatomic undercuts, limited means of retention, mobility of soft tissue margins, and the weight of the prosthesis.. Use of skin adhesives may be precluded because of the presence of persistent moisture and saliva.. The maxillofacial rehabilitation, including the design and fabrication of a 2-piece silicone prosthesis retained by the teeth, of a patient with a large facial defect as a result of treatment for an arteriovenous malformation is described.

    Article Title: Microfluidic device and method for concentrating sample containing cells or viruses and lysing cells or viruses, and method of producing the microfluidic device
    Article Snippet: .. Dow Corning Primer 1205 was spin coated (500 revolutions per minute (“rpm”), 5 seconds (s); 700 rpm, 10 s) on the silicon wafer as an adhesive, a cation exchange membrane containing —SO3—Na+ group is bonded on the silicon wafer using a roller, and then Dow Corning Primer 1205 was spin coated thereon (500 rpm, 5 s; 1500 rpm, 10 s) to form a first adhesive layer and a second adhesive layer on the cation exchange membrane. .. The Dow Corning Primer 1205 includes a low viscosity, such as water does, at room temperature and has no adhesiveness.

    Modification:

    Article Title: Maxillofacial rehabilitation of a large facial defect resulting from an arteriovenous malformation utilizing a two-piece prosthesis.
    Article Snippet: Large facial defects involving the oral cavity can be difficult to restore prosthetically because of a lack of anatomic undercuts, limited means of retention, mobility of soft tissue margins, and the weight of the prosthesis.. Use of skin adhesives may be precluded because of the presence of persistent moisture and saliva.. The maxillofacial rehabilitation, including the design and fabrication of a 2-piece silicone prosthesis retained by the teeth, of a patient with a large facial defect as a result of treatment for an arteriovenous malformation is described.

    Membrane:

    Article Title: Microfluidic device and method for concentrating sample containing cells or viruses and lysing cells or viruses, and method of producing the microfluidic device
    Article Snippet: .. Dow Corning Primer 1205 was spin coated (500 revolutions per minute (“rpm”), 5 seconds (s); 700 rpm, 10 s) on the silicon wafer as an adhesive, a cation exchange membrane containing —SO3—Na+ group is bonded on the silicon wafer using a roller, and then Dow Corning Primer 1205 was spin coated thereon (500 rpm, 5 s; 1500 rpm, 10 s) to form a first adhesive layer and a second adhesive layer on the cation exchange membrane. .. The Dow Corning Primer 1205 includes a low viscosity, such as water does, at room temperature and has no adhesiveness.



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    Image Search Results


    CircIL21R served as a sponge for miR-1205. A: Venn diagram showing the intersection of the circIntercome and circBANK databases that predicted the two potential target miRNAs of circIL21R. B: Diagram of the binding site of circIL21R to miR-1205. C, D: qPCR assays confirmed that the overexpression of miR-1205 in ME180 cells and the knockdown of miR-1205 in SiHa cells resulted in a more significant change in miR-1205 expression than in miR-1244 expression. E: qPCR assays showed that the relative expression of miR-1205 was lower in tumor tissue than in adjacent normal tissue. F, G: qPCR assays illustrated that miR-1205-inhibitor treatment (ME180 and C33A) and miR-1205-mimic treatment (Caski and SiHa) reversely regulated the expression of circIL21R. H-K: Luciferase reporter gene assays showed that miR-1205 inhibitor treatment (ME180 and C33A) and miR-1205 mimic treatment (Caski and SiHa) altered the luciferase activity of circIL21R. L, M: RIP assays demonstrated that miR-1205 and circIL21R were effectively pulled down by anti-AGO2 antibodies compared with IgG and were enriched after mimic treatment with miR-1205 in SiHa cells. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: American Journal of Cancer Research

    Article Title: Circular RNA circIL21R promotes cervical cancer cells proliferation and migration by sponging the miR-1205/PTBP1 axis

    doi: 10.62347/XYCC4213

    Figure Lengend Snippet: CircIL21R served as a sponge for miR-1205. A: Venn diagram showing the intersection of the circIntercome and circBANK databases that predicted the two potential target miRNAs of circIL21R. B: Diagram of the binding site of circIL21R to miR-1205. C, D: qPCR assays confirmed that the overexpression of miR-1205 in ME180 cells and the knockdown of miR-1205 in SiHa cells resulted in a more significant change in miR-1205 expression than in miR-1244 expression. E: qPCR assays showed that the relative expression of miR-1205 was lower in tumor tissue than in adjacent normal tissue. F, G: qPCR assays illustrated that miR-1205-inhibitor treatment (ME180 and C33A) and miR-1205-mimic treatment (Caski and SiHa) reversely regulated the expression of circIL21R. H-K: Luciferase reporter gene assays showed that miR-1205 inhibitor treatment (ME180 and C33A) and miR-1205 mimic treatment (Caski and SiHa) altered the luciferase activity of circIL21R. L, M: RIP assays demonstrated that miR-1205 and circIL21R were effectively pulled down by anti-AGO2 antibodies compared with IgG and were enriched after mimic treatment with miR-1205 in SiHa cells. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: The primers of miR-1205 were purchased from Thermo Fisher Scientific (Primer ID: Hs00508322_CE).

    Techniques: Binding Assay, Over Expression, Expressing, Luciferase, Activity Assay

    Overexpression of circIL21R promoted cervical cancer progression via miR-1205. A, B: MTS assays showed that the increase in absorbance caused by circIL21R overexpression decreased after miR-1205 mimic treatment in ME180 and C33A cells. C-F: EDU assays confirmed that miR-1205 mimic treatment reversed the proliferation-promoting effect of circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. G, H: Transwell assays confirmed that miR-1205 mimic treatment offset the enhanced invasion capacity caused by circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: American Journal of Cancer Research

    Article Title: Circular RNA circIL21R promotes cervical cancer cells proliferation and migration by sponging the miR-1205/PTBP1 axis

    doi: 10.62347/XYCC4213

    Figure Lengend Snippet: Overexpression of circIL21R promoted cervical cancer progression via miR-1205. A, B: MTS assays showed that the increase in absorbance caused by circIL21R overexpression decreased after miR-1205 mimic treatment in ME180 and C33A cells. C-F: EDU assays confirmed that miR-1205 mimic treatment reversed the proliferation-promoting effect of circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. G, H: Transwell assays confirmed that miR-1205 mimic treatment offset the enhanced invasion capacity caused by circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: The primers of miR-1205 were purchased from Thermo Fisher Scientific (Primer ID: Hs00508322_CE).

    Techniques: Over Expression

    PTBP1 is a downstream target oncogene of miR-1205. A: Venn diagram illustrating an intersection of four databases that predicted the two potential target oncogenes of miR-1205. B, C: qPCR assays confirmed that miR-1205-inhibitor treatment in ME180 cells and miR-1205-mimic treatment in SiHa cells resulted in a more significant change in PTBP1 expression than in SBNO1 expression. D: Diagram of the binding site of miR-1205 in PTBP1. E: The expression of PTBP1 in cervical cancer tissue was greater than that in normal tissue based on the TCGA dataset. F: Kaplan-Meier survival analysis revealed that cervical cancer patients with lower PTBP1 expression had better survival outcomes than those with higher PTBP1 expression. G: ROC curve analysis indicated that PTBP1 was an effective diagnostic biomarker of cervical cancer. H-K: Luciferase reporter gene assays demonstrated that miR-1205 inhibitor treatment (ME180 and C33A) and miR-1205 mimic treatment (Caski and SiHa) regulated the luciferase activity of PTBP1. L: Western blotting showing the expression of PTBP1 after miR-1205 inhibition in ME180 cells and mimic treatment in SiHa cells. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: American Journal of Cancer Research

    Article Title: Circular RNA circIL21R promotes cervical cancer cells proliferation and migration by sponging the miR-1205/PTBP1 axis

    doi: 10.62347/XYCC4213

    Figure Lengend Snippet: PTBP1 is a downstream target oncogene of miR-1205. A: Venn diagram illustrating an intersection of four databases that predicted the two potential target oncogenes of miR-1205. B, C: qPCR assays confirmed that miR-1205-inhibitor treatment in ME180 cells and miR-1205-mimic treatment in SiHa cells resulted in a more significant change in PTBP1 expression than in SBNO1 expression. D: Diagram of the binding site of miR-1205 in PTBP1. E: The expression of PTBP1 in cervical cancer tissue was greater than that in normal tissue based on the TCGA dataset. F: Kaplan-Meier survival analysis revealed that cervical cancer patients with lower PTBP1 expression had better survival outcomes than those with higher PTBP1 expression. G: ROC curve analysis indicated that PTBP1 was an effective diagnostic biomarker of cervical cancer. H-K: Luciferase reporter gene assays demonstrated that miR-1205 inhibitor treatment (ME180 and C33A) and miR-1205 mimic treatment (Caski and SiHa) regulated the luciferase activity of PTBP1. L: Western blotting showing the expression of PTBP1 after miR-1205 inhibition in ME180 cells and mimic treatment in SiHa cells. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: The primers of miR-1205 were purchased from Thermo Fisher Scientific (Primer ID: Hs00508322_CE).

    Techniques: Expressing, Binding Assay, Diagnostic Assay, Biomarker Assay, Luciferase, Activity Assay, Western Blot, Inhibition

    CircIL21R regulated PTBP1 expression via miR-1205. A-C: Western blotting and qPCR assays revealed that the increase in PTBP1 expression induced by circIL21R overexpression was reversed after miR-1205 mimic treatment in ME180 and C33A cells. D, E: MTS assays demonstrated that the increase in viability of ME180 and C33A cells promoted by circIL21R overexpression was reversed after PTBP1 knockdown. F-H: The EdU assay revealed that the increased proliferation of ME180 and C33A cells caused by circIL21R overexpression was reversed after PTBP1 knockdown. Scale bar = 100 µm. I, J: Transwell assays revealed that PTBP1 knockdown inhibited the enhanced tumor invasion capacity induced by circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: American Journal of Cancer Research

    Article Title: Circular RNA circIL21R promotes cervical cancer cells proliferation and migration by sponging the miR-1205/PTBP1 axis

    doi: 10.62347/XYCC4213

    Figure Lengend Snippet: CircIL21R regulated PTBP1 expression via miR-1205. A-C: Western blotting and qPCR assays revealed that the increase in PTBP1 expression induced by circIL21R overexpression was reversed after miR-1205 mimic treatment in ME180 and C33A cells. D, E: MTS assays demonstrated that the increase in viability of ME180 and C33A cells promoted by circIL21R overexpression was reversed after PTBP1 knockdown. F-H: The EdU assay revealed that the increased proliferation of ME180 and C33A cells caused by circIL21R overexpression was reversed after PTBP1 knockdown. Scale bar = 100 µm. I, J: Transwell assays revealed that PTBP1 knockdown inhibited the enhanced tumor invasion capacity induced by circIL21R overexpression in ME180 and C33A cells. Scale bar = 100 µm. All the data are expressed as the means ± SDs (of three independent experiments). *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: The primers of miR-1205 were purchased from Thermo Fisher Scientific (Primer ID: Hs00508322_CE).

    Techniques: Expressing, Western Blot, Over Expression, EdU Assay

    Groups division according to the presence or not of scratches, primer and adhesive

    Journal: Journal of Applied Oral Science

    Article Title: Bond strength between acrylic resin and maxillofacial silicone

    doi: 10.1590/S1678-77572012000600010

    Figure Lengend Snippet: Groups division according to the presence or not of scratches, primer and adhesive

    Article Snippet: Therefore, a 30-minute period was allowed after the Dow Corning 1205 Primer (Dow Corning Corporation, Midland, MI, USA) or Sofreliner Primer S (Tokuyama Corp., Taitou-ku, Tokyo, Japan) application so that the primer could react with the resin surface.

    Techniques: